OMIA:001567-9986 : Hypercholesterolaemia, Kurosawa and Kusanagi in Oryctolagus cuniculus (rabbit)

Categories: Homeostasis / metabolism phene

Links to MONDO diseases: No links.

Mendelian trait/disorder: yes

Mode of inheritance: Autosomal recessive

Considered a defect: yes

Key variant known: no

Genetic engineering: Unknown
Have human generated variants been created, e.g. through genetic engineering and gene editing

Cite this entry

Nicholas, F. W., Tammen, I., & Sydney Informatics Hub. (2011). OMIA:001567-9986: Online Mendelian Inheritance in Animals (OMIA) [dataset]. https://omia.org/. https://doi.org/10.25910/2AMR-PV70

References

Note: the references are listed in reverse chronological order (from the most recent year to the earliest year), and alphabetically by first author within a year.

2011 Hayashi, S., Obara, S., Murakawa, M., Hazama, A., Kusanagi, M., Katsuda, S. :
To what extent does aortic pulse wave velocity estimate early atherosclerosis in Kurosawa and Kusanagi-hypercholesterolemic rabbits? Hypertens Res 34:559-64, 2011. Pubmed reference: 21228784. DOI: 10.1038/hr.2010.273.
2004 Yoshie, F., Iizuka, A., Komatsu, Y., Matsumoto, A., Itakura, H., Kondo, K. :
Effects of Dai-saiko-to (Da-Chai-Hu-Tang) on plasma lipids and atherosclerotic lesions in female heterozygous heritable Kurosawa and Kusanagi-hypercholesterolemic (KHC) rabbits. Pharmacol Res 50:223-30, 2004. Pubmed reference: 15225663. DOI: 10.1016/j.phrs.2004.02.003.
1995 Kurosawa, T., Kusanagi, M., Yamasaki, Y., Senga, Y., Yamamoto, T. :
New mutant rabbit strain with hypercholesterolemia and atherosclerotic lesions produced by serial inbreeding. Lab Anim Sci 45:385-92, 1995. Pubmed reference: 7474877.

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  • Created by Frank Nicholas on 27 May 2011