OMIA:003066-8128 : Genetically engineered gene knockout or knockin, generic in Oreochromis niloticus (Nile tilapia)

In other species: grass carp , Japanese medaka , pig , golden hamster , turquoise killifish

Categories: Cellular phene

Single-gene trait/disorder: unknown

Disease-related: unknown

Cross-species summary: Gene knockout and knockin models comprise genetically engineered animals in which specific genes have been inactivated or deleted or added. These models provide a valuable tool for investigating gene function, disease mechanisms, and biological processes. See also gene specific entries (by doing an exhaustive search for GMO) and/or entires for OMIA:002665  Genetically engineered humanised animal.

Genetic engineering: Yes - variants have been created artificially, e.g. by genetic engineering or gene editing
Have human generated variants been created, e.g. through genetic engineering and gene editing

Associated gene:

Symbol Description Species Chr Location OMIA gene details page Other Links
ptch2 patched 2 Oreochromis niloticus LG18 NC_031982.2 (15436476..15408793) ptch2 Ensembl, NCBI gene

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Cite this entry

Nicholas, F. W., Tammen, I., & Sydney Informatics Hub. (2026). OMIA:003066-8128: Online Mendelian Inheritance in Animals (OMIA) [dataset]. https://omia.org/. https://doi.org/10.25910/2AMR-PV70

Reference

2026 Zhao, C., Liu, X., Peng, X., Chen, Y., Peng, S., Liu, L., Wang, D., Wei, J. :
Ptch2 deficiency triggers lipoma formation and adipogenic transcriptome reprogramming in Nile tilapia (Oreochromis niloticus). Animals (Basel) 16:405, 2026. Pubmed reference: 41681386. DOI: 10.3390/ani16030405.

Edit History


  • Created by Imke Tammen on 25 Jul 2026